Autism and Metabolic Cytopathy
Mehmet Emin Ceylan, Ayse Fulya Maner*, Ahmet Turkcan, Agah Aydin
Identifiers and Pagination:Year: 2011
First Page: 49
Last Page: 50
Publisher ID: TONIJ-5-49
Article History:Received Date: 7/2/2011
Revision Received Date: 6/4/2011
Acceptance Date: 11/4/2011
Electronic publication date: 5/8/2011
Collection year: 2011
open-access license: This is an open access article licensed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted, non-commercial use, distribution and reproduction in any medium, provided the work is properly cited.
Letter to the editor: Autism is a wide spectrum disorder and a lot of factors play role in the etiology. Autism may accompany some genetic disorders such as fragile X, tuberosclerosis, neurofibromatosis and phenylketonuria . However, the absence of sufficient evidence on the etiological roles of environmental, neuroanatomical and biochemical factors has shifted the direction of research to genetics and cytology .