RESEARCH ARTICLE


Autism and Metabolic Cytopathy



Mehmet Emin Ceylan, Ayse Fulya Maner*, Ahmet Turkcan, Agah Aydin
Bakirkoy Research and Training Hospital for Psychiatry, Neurology and Neurosurgery, Istanbul, Turkey


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© Ceylan et al; Licensee Bentham Open

open-access license: This is an open access article licensed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted, non-commercial use, distribution and reproduction in any medium, provided the work is properly cited.

* Address correspondence to this author at the Bakirkoy Research and Training Hospital for Psychiatry, Neurology and Neurosurgery, Istanbul, Turkey; Tel: 00905322414102; Fax: 00901255729595; E-mail: fmaner@ttmail.com


Abstract

Letter to the editor: Autism is a wide spectrum disorder and a lot of factors play role in the etiology. Autism may accompany some genetic disorders such as fragile X, tuberosclerosis, neurofibromatosis and phenylketonuria [1]. However, the absence of sufficient evidence on the etiological roles of environmental, neuroanatomical and biochemical factors has shifted the direction of research to genetics and cytology [2].

Keywords: Autism, genetics, metabolic cytopathy.